BIOGRAPHICAL SKETCH |
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| NAME: Johnson Haynes, Jr., M.D. |
POSITION
TITLE: Professor of Medicine |
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| EDUCATION/TRAINING: | ||||
| INSTITUTION AND LOCATION: | DEGREE: |
YEAR(s): |
FIELD OF STUDY: |
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| Tuskegee Institute, Tuskegee, Alabama | B.S. |
1975 |
Biology | |
| University of South Alabama College of Medicine | M.D. |
1980 |
Medicine | |
| University of South Alabama Medical Center | 1980-1983 |
Residency | ||
| University of South Alabama Medical Center | 1983-1984 |
Chief Medical Resident | ||
| University of South Alabama Medical Center | 1984-1986 |
Pulmonary Medicine/Research Fellow | ||
| University of Colorado | 1986-1988 |
Pulmonary Research Fellow | ||
PROFESSIONAL EXPERIENCE
1988-1992 Assistant Professor, Department of Medicine, University of South Alabama College of Medicine, Mobile, AL
1992-1995 Associate Professor, Department of Medicine, University of South Alabama College of Medicine, Mobile, AL
1993-present Associate Director of Clinical Programs, University of South Alabama, Comprehensive Sickle Cell Center, Mobile, AL
1995-present Professor of Medicine, Department of Medicine, University of South Alabama College of Medicine, Mobile, AL
HONORS:
National Research Service Award
American Lung Association Fellowship Recipient
Alpha Kappa Mu Honor Society
Who's Who in American Colleges and Universities
Victor Benator Award for Teaching Excellence
Alpha Omega Honor Medical Society
Fellow, American College of Physicians
Fellow, American College of Chest Physicians
PUBLICATIONS:
Haynes J, Seibert A, Shah A, and Taylor A. Normal vs sickle red blood cells: hemodynamic and permeability characteristics in reperfusion lung injury. JAAMP 1(3):62-66, 1990.
Kirkpatrick MB, Haynes J, and Bass J. Results of bronchoscopically-obtained lower airway cultures from adult sickle cell disease patients with acute chest syndrome. Am J Med 90:206-210, 1991.
Seibert A, Taylor AE, Bass J, and Haynes J. Hemoglobin potentiates oxidant injury in isolated rat lungs. Am J Physiol 260 (Heart Circ Physiol 29):H1980-H1984, 1991.
Seibert AF, Taylor A, Thompson WJ, Wilborn WH, Barnard J, and Haynes J. Reversal of increased microvascular permeability associated with ischemia-reperfusion: role of cAMP. J Appl Physiol 72(1):389-395, 1992.
Adkins WK, Barnard JW, May S, Seibert A, Haynes J, and Taylor AE. Compounds which increase cyclic-AMP prevent ischemia-reperfusion pulmonary capillary injury. J Appl Physiol 72(2):492-497, 1992.
Haynes J, Taylor AE, Dixon D, and Voelkel N. Microvascular hemodynamics in the sickle red blood cell perfused isolated rat lung. Am J Physiol 264(Heart Circ Physiol 33):H484-H489, 1993.
Haynes J and Bass J. Sickle cell lung disease. Pulmonary Critical Care Update, Lesson 20, Volume 8, 1993.
Haynes J and Kirkpatrick M. The acute chest syndrome of sickle cell disease. Am J Med Sci 305(5):326-330, 1993.
Seibert AF, Haynes J, and Taylor A. Ischemia-reperfusion injury in the isolated rat lung: role of flow and endogenous leukocytes. Am Rev Respir Dis 147:270-275, 1993.
Castro OL, Chicoye LS, Greenberg J, Haynes J, and Peterson KR. Brighter horizons for sickle cell disease. Patient Care 28(8):26-44, 1994.
Kirkpatrick MB and Haynes J. Sickle cell disease and the pulmonary circulation. Sem in the Respir Crit Care Med 15(6):473-481, 1994.
Haynes J, Obiako B, Thompson J, and Downey J. Adenosine-induced vasodilation: receptor characterization in pulmonary circulation. Am J Physiol 268(Heart Circ Physiol 37):H1862-H1868, 1995.
Haynes J, Killilea DW, Peterson PD, and Thompson WJ. Erythro-9-(2-hydroxy-3-nonyl) adenine (EHNA) inhibits cGMP-stimulated phosphodiesterase (PEDE2) to reverse hypoxic pulmonary vasoconstriction in the perfused rat lung. J Pet 276:752-757, 1996.
Haynes J, Manci E, Voelkel N. The lung in sickle cell disease. In: The Sickle Hemoglobinopathies: Science and Medicine. Edited by SH Embury, RP Hebbel, N Mohandas and M Steinberg. Raven Press, 1994.
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This page last updated 6-25-98.
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