BIOGRAPHICAL SKETCH

NAME:

Johnson Haynes, Jr., M.D.

POSITION TITLE:

Professor of Medicine

EDUCATION/TRAINING:
INSTITUTION AND LOCATION:

DEGREE:

YEAR(s):

FIELD OF STUDY:

Tuskegee Institute, Tuskegee, Alabama

B.S.

1975

Biology
University of South Alabama College of Medicine

M.D.

1980

Medicine
University of South Alabama Medical Center  

1980-1983

Residency
University of South Alabama Medical Center  

1983-1984

Chief Medical Resident
University of South Alabama Medical Center  

1984-1986

Pulmonary Medicine/Research Fellow
University of Colorado  

1986-1988

Pulmonary Research Fellow

PROFESSIONAL EXPERIENCE

1988-1992         Assistant Professor, Department of Medicine, University of South Alabama College of Medicine, Mobile, AL

1992-1995         Associate Professor, Department of Medicine, University of South Alabama College of Medicine, Mobile, AL

1993-present     Associate Director of Clinical Programs, University of South Alabama, Comprehensive Sickle Cell Center, Mobile, AL

1995-present     Professor of Medicine, Department of Medicine, University of South Alabama College of Medicine, Mobile, AL

HONORS:

National Research Service Award

American Lung Association Fellowship Recipient

Alpha Kappa Mu Honor Society

Who's Who in American Colleges and Universities

Victor Benator Award for Teaching Excellence

Alpha Omega Honor Medical Society

Fellow, American College of Physicians

Fellow, American College of Chest Physicians

PUBLICATIONS:

Haynes J, Seibert A, Shah A, and Taylor A. Normal vs sickle red blood cells: hemodynamic and permeability characteristics in reperfusion lung injury. JAAMP 1(3):62-66, 1990.

Kirkpatrick MB, Haynes J, and Bass J. Results of bronchoscopically-obtained lower airway cultures from adult sickle cell disease patients with acute chest syndrome. Am J Med 90:206-210, 1991.

Seibert A, Taylor AE, Bass J, and Haynes J. Hemoglobin potentiates oxidant injury in isolated rat lungs. Am J Physiol 260 (Heart Circ Physiol 29):H1980-H1984, 1991.

Seibert AF, Taylor A, Thompson WJ, Wilborn WH, Barnard J, and Haynes J. Reversal of increased microvascular permeability associated with ischemia-reperfusion: role of cAMP. J Appl Physiol 72(1):389-395, 1992.

Adkins WK, Barnard JW, May S, Seibert A, Haynes J, and Taylor AE. Compounds which increase cyclic-AMP prevent ischemia-reperfusion pulmonary capillary injury. J Appl Physiol 72(2):492-497, 1992.

Haynes J, Taylor AE, Dixon D, and Voelkel N. Microvascular hemodynamics in the sickle red blood cell perfused isolated rat lung. Am J Physiol 264(Heart Circ Physiol 33):H484-H489, 1993.

Haynes J and Bass J. Sickle cell lung disease. Pulmonary Critical Care Update, Lesson 20, Volume 8, 1993.

Haynes J and Kirkpatrick M. The acute chest syndrome of sickle cell disease. Am J Med Sci 305(5):326-330, 1993.

Seibert AF, Haynes J, and Taylor A. Ischemia-reperfusion injury in the isolated rat lung: role of flow and endogenous leukocytes. Am Rev Respir Dis 147:270-275, 1993.

Castro OL, Chicoye LS, Greenberg J, Haynes J, and Peterson KR. Brighter horizons for sickle cell disease. Patient Care 28(8):26-44, 1994.

Kirkpatrick MB and Haynes J. Sickle cell disease and the pulmonary circulation. Sem in the Respir Crit Care Med 15(6):473-481, 1994.

Haynes J, Obiako B, Thompson J, and Downey J. Adenosine-induced vasodilation: receptor characterization in pulmonary circulation. Am J Physiol 268(Heart Circ Physiol 37):H1862-H1868, 1995.

Haynes J, Killilea DW, Peterson PD, and Thompson WJ. Erythro-9-(2-hydroxy-3-nonyl) adenine (EHNA) inhibits cGMP-stimulated phosphodiesterase (PEDE2) to reverse hypoxic pulmonary vasoconstriction in the perfused rat lung. J Pet 276:752-757, 1996.

Haynes J, Manci E, Voelkel N. The lung in sickle cell disease. In: The Sickle Hemoglobinopathies: Science and Medicine. Edited by SH Embury, RP Hebbel, N Mohandas and M Steinberg. Raven Press, 1994.

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